If you reside in an apartment or a house or simply would like to keep your home free of EMFs, there are a number options to limit your exposure. One of the most effective is to reduce the usage of your electronic devices. You could also consider EMF blocker paint to block EMF radiation from entering your house. emf blocking to protect your home from EMF radiations is to use a RF shielding canopy. It's a kind of net that has EMF shielding. It's used to block EMFs from entering rooms. Another option is to get your home equipped with a conductive enclosure. These enclosures are known as Faraday cages.
block emf have shown that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to involve down-regulation of cancer stem cells. emf blockers could be the reason for the long-term response seen in patients with advanced HCC. However, the reason behind AM RF EMF's effect in patients with cancer is not yet clear.
The effects on the effects of AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumours were separated in three different groups. One group did not have exposure to RF EMF. Second group members were exposed RF EMF at the same frequency to the frequency used by humans. In the third, they were subjected the RF EMF in HCC-specific frequencies. The effects of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of tumour shrinkage.
The mechanism of tumour-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused by CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have wider implications in the treatment and diagnosis of various cancers.
The tumours in the controls were never exposed RF EMF, and were fed a normal mouse diet. The tumours in the HCCMF group were injected with Huh7 cells after they were five-seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumours that were treated using RCF showed no signs of shrinkage. The difference was highly significant. The tumours treated with RCF had necrosis, which is typical when tumors are that are exposed to RCF. There is a possibility that the necrosis was caused by a lack of oxygen in the larger tumors.
In conclusion, the findings indicate the fact that AM EMF exhibits anti-cancer properties in vitro as well as in vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF produces these effects through CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the development of HCC tumors in living tissue.