No matter if you live in an apartment or house or just would like to keep your home free from EMFs, there are a number options to limit your exposure. One of the most straightforward is to restrict the usage of your electronic devices. You could also consider EMF blocker paint to block EMF radiation from reaching your home. emf blocker to shield your home against EMF radiation is to put up an RF shielding canopy. This is a cloth made of net which contains EMF shielding. It is utilized to block EMFs from entering a room. Another option is to have your home equipped with a conductive enclosure. These enclosures are known as Faraday cages.
A number of studies have proven that the non-ionizing RF EMF produces anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation of cancer stem cells. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in patients with cancer is not evident.
Effects from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumours were separated in three different groups. First, the group that was unaffected RF EMF. Second group members were exposed RF EMF at the same frequency to that of humans. Third group members were exposed the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results revealed that tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage in the tumour.
emf blocker behind tumour-specific AM RF EMF could be driven by the fact that cancer cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled through CACNA1H, a protein which mediates tumour-specific Ca2+ influx. emf blockers suggest that CACNA1H could have more broader implications in the diagnosis and treatment of various cancers.
The tumours of the control group were not exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors of those in the HCCMF group were injected with Huh7 cells after they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumours in the three groups also displayed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in size of the tumor after eight weeks. However, the tumors which were treated by RCF did not show any signs of shrinkage. The difference was highly significant. The tumours treated with RCF showed necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that this necrosis is caused by the lack of oxygen in the larger cancers.
In conclusion, the findings show the fact that AM EMF exhibits anticancer effects in vitro and in the vivo. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. The possibility is that the AM EMF produces these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting effect on the development of HCC tumors in the vivo.