If you reside in an apartment or a house or just want to keep your home free of EMFs, there are a number options to limit your exposure. One of the easiest is to reduce the use of electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your home. Another method to protect your house against EMF radiations is to put up a RF shielding canopy. It's a kind of net that has EMF shielding. block emf 's used to stop EMFs from entering a room. Another option is to get your home fitted with a conductive enclosure. These enclosures are known as Faraday cages.
Several studies have shown how the EMF that is not ionized has anti-proliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. This could be the reason for the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM EMF's effects on cancer patients isn't evident.
Effects on the effects of AM RF EMFs on HCC tumour growth in vivo was studied in mice. The tumors were classified in three different groups. One group did not have exposure to RF EMF. Second group members were exposed RF EMF at frequencies similar to that used in humans. Third group members were exposed RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumours treated with HCCMF had significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF might be driven by the fact that tumour cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications for the treatment and diagnosis of various cancers.
The tumors in the control group were not exposed to EMF from RF, and fed a normal mouse diet. The tumors of those in the HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were then killed in cases of excessive burden.
The tumors of the three groups also displayed distinct growth curves. The HCCMF-treated tumors saw a significant decrease in tumour size after eight weeks. However, emf blocker treated with RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis that is common when tumors are exposed to RCF. There is a possibility that the necrosis was caused by the lack of oxygen in the larger tumors.
In emf blockers , the results suggest the fact that AM EMF is a powerful source of anticancer effects in vitro and in live. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF produces these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting impact on the development of HCC tumours in vivo.